- 1. Collapsing glomerulopathy (CG)—high-dose pamidronate
- 2. Thrombotic microangiopathy (TMA)—bevacizumab; anti-vascular endothelial growth factor (anti-VEGF) agent
- 3. Severe acute tubular necrosis (ATN)—ifosfamide
- 4. Minimal change disease (MCD) and diffuse podocytopathy—doxorubicin
- 5. Lupus-like glomerulonephritis—ipilimumab
- References
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1.
Perazella MA, Markowitz GS. Bisphosphonate nephrotoxicity. Kidney Int 2008; 74:1385–1393. doi: 10.1038/ki.2008.356
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2.
Izzedine H, et al. Thrombotic microangiopathy and anti-VEGF agents. Nephrol Dial Transplant 2007; 22:1481–1482. doi: 10.1093/ndt/gfl565
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3.
Nissim I, et al. Ifosfamide-induced nephrotoxicity: Mechanism and prevention. Cancer Res 2006; 66:7824–7831. doi: 10.1158/0008-5472.CAN-06-1043
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4.
Bertani T, et al. Steroids and adriamycin nephrosis. Appl Pathol 1984; 2:32–38. PMID: 6525317; https://www.researchgate.net/publication/16671624_Steroids_and_Adriamycin_nephrosis
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5.
Fadel F, et al. Anti-CTLA4 antibody-induced lupus nephritis. N Engl J Med 2009; 361:211–212. doi: 10.1056/NEJMc0904283
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